SRT2104 extends survival of male mice on a standard diet and preserves bone and muscle mass

نویسندگان

  • Evi M Mercken
  • Sarah J Mitchell
  • Alejandro Martin-Montalvo
  • Robin K Minor
  • Maria Almeida
  • Ana P Gomes
  • Morten Scheibye-Knudsen
  • Hector H Palacios
  • Jordan J Licata
  • Yongqing Zhang
  • Kevin G Becker
  • Husam Khraiwesh
  • José A González-Reyes
  • José M Villalba
  • Joseph A Baur
  • Peter Elliott
  • Christoph Westphal
  • George P Vlasuk
  • James L Ellis
  • David A Sinclair
  • Michel Bernier
  • Rafael de Cabo
چکیده

Increased expression of SIRT1 extends the lifespan of lower organisms and delays the onset of age-related diseases in mammals. Here, we show that SRT2104, a synthetic small molecule activator of SIRT1, extends both mean and maximal lifespan of mice fed a standard diet. This is accompanied by improvements in health, including enhanced motor coordination, performance, bone mineral density, and insulin sensitivity associated with higher mitochondrial content and decreased inflammation. Short-term SRT2104 treatment preserves bone and muscle mass in an experimental model of atrophy. These results demonstrate it is possible to design a small molecule that can slow aging and delay multiple age-related diseases in mammals, supporting the therapeutic potential of SIRT1 activators in humans.

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عنوان ژورنال:

دوره 13  شماره 

صفحات  -

تاریخ انتشار 2014